Many drugs currently used in anticancer therapies, act by activating cytotoxic death by apoptosis. This death mechanism is often accompanied by mitochondrial events (intrinsic apoptosis) or the activation of death receptors (extrinsic pathway)
Pellerito, C., Prinzivalli, C., Casella, G., Fiore, T., Pellerito, O., Giuliano, M., et al. (2010). Bu2Sn(N-acetyl-L-cysteinate) antitumor activity on HepG2 cells. In Tenth Workshop on Pharmaco-Biometallics workshop (pp. 48-48).
Bu2Sn(N-acetyl-L-cysteinate) antitumor activity on HepG2 cells
PELLERITO, Claudia;PRINZIVALLI, Cristina;CASELLA, Girolamo;FIORE, Tiziana;PELLERITO, Ornella;GIULIANO, Michela;SCOPELLITI, Michelangelo;PELLERITO, Lorenzo
2010-01-01
Abstract
Many drugs currently used in anticancer therapies, act by activating cytotoxic death by apoptosis. This death mechanism is often accompanied by mitochondrial events (intrinsic apoptosis) or the activation of death receptors (extrinsic pathway)
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