Inflammation may have a pathogenic role in the progression of nonalcoholic fatty liver disease (NAFLD); by contrast, the role of anti-inflammatory molecules has not been addressed. Low circulating levels of the anti-inflammatory molecule IGF-I have been described in subjects with NAFLD.
Hribal, M., Procopio, T., Petta, S., Sciacqua, A., Grimaudo, S., Pipitone, R., et al. (2013). Insulin-like growth factor-I, inflammatory proteins, and fibrosis in subjects with nonalcoholic fatty liver disease. THE JOURNAL OF CLINICAL ENDOCRINOLOGY AND METABOLISM, 98(2), E304-E308 [10.1210/jc.2012-3290].
Insulin-like growth factor-I, inflammatory proteins, and fibrosis in subjects with nonalcoholic fatty liver disease
PETTA, Salvatore;GRIMAUDO, Stefania;PIPITONE, Rosaria Maria;
2013-01-01
Abstract
Inflammation may have a pathogenic role in the progression of nonalcoholic fatty liver disease (NAFLD); by contrast, the role of anti-inflammatory molecules has not been addressed. Low circulating levels of the anti-inflammatory molecule IGF-I have been described in subjects with NAFLD.
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